Forced expression of the cell cycle inhibitor p57Kip2 in cardiomyocytes attenuates ischemia-reperfusion injury in the mouse heart

Table 1 Genotype analysis of offspring resulting from R26loxp-TAp57^k/+;Mlc2v-cre^k/+crosses.

The R26loxp-TAp57^k/+;Mlc2v-cre^k/+genotype is represented in the expected Mendelian ratio in the offspring. However, mice with p57^KIP2 cardiac-specific overexpression that are homozygous for the R26p57 allele (R26loxp-TAp57^k/k;Mlc2v-cre^k/+) are significantly under-represented in the offspring (p < 0.01 by Chi-square and n = 203)

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